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Nociceptor
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==Terminology== [[Nociception]] and pain are usually evoked only by pressures and temperatures that are potentially damaging to tissues. This barrier or [[threshold of pain|threshold]] contrasts with the more sensitive visual, auditory, olfactory, taste, and somatosensory responses to stimuli. The experience of pain is individualistic and can be suppressed by stress or exacerbated by anticipation. Simple activation of a nociceptor does not always lead to perceived pain, because the latter also depends on the frequency of the [[action potentials]], integration of pre- and postsynaptic signals, and influences from higher or central processes.<ref name="Nociceptors: the sensors of the pai">{{cite journal |last1=Dubin |first1=AE |last2=Patapoutian |first2=A |title=Nociceptors: the sensors of the pain pathway. |journal=The Journal of Clinical Investigation |date=November 2010 |volume=120 |issue=11 |pages=3760β72 |doi=10.1172/JCI42843 |pmid=21041958|pmc=2964977 }}</ref>
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