Editing Staphylococcus aureus (section)

=== Enzymes ===
''Staphylococcus aureus'' produces various enzymes such as [[coagulase]] (bound and free coagulases) which facilitates the conversion of fibrinogen to fibrin to cause clots which is important in skin infections.<ref>{{cite journal | vauthors = Cheung GY, Bae JS, Otto M | title = Pathogenicity and virulence of ''Staphylococcus aureus'' | journal = Virulence | volume = 12 | issue = 1 | pages = 547–569 | date = December 2021 | pmid = 33522395 | pmc = 7872022 | doi = 10.1080/21505594.2021.1878688 }}</ref> [[Hyaluronidase]] (also known as spreading factor) breaks down [[hyaluronan|hyaluronic acid]] and helps in spreading it. [[Deoxyribonuclease]], which breaks down the DNA, protects ''S. aureus'' from [[Neutrophil extracellular traps|neutrophil extracellular trap]]-mediated killing.<ref>{{cite journal | vauthors = Berends ET, Horswill AR, Haste NM, Monestier M, Nizet V, von Köckritz-Blickwede M | title = Nuclease expression by ''Staphylococcus aureus'' facilitates escape from neutrophil extracellular traps | language = english | journal = Journal of Innate Immunity | volume = 2 | issue = 6 | pages = 576–586 | date = 2010 | pmid = 20829609 | pmc = 2982853 | doi = 10.1159/000319909 }}</ref><ref>{{cite journal | vauthors = Monteith AJ, Miller JM, Maxwell CN, Chazin WJ, Skaar EP | title = Neutrophil extracellular traps enhance macrophage killing of bacterial pathogens | language = EN | journal = Science Advances | volume = 7 | issue = 37 | pages = eabj2101 | date = September 2021 | pmid = 34516771 | pmc = 8442908 | doi = 10.1126/sciadv.abj2101 | bibcode = 2021SciA....7.2101M | doi-access = free }}</ref> ''S. aureus'' also produces [[lipase]] to digest lipids, [[staphylokinase]] to dissolve fibrin and aid in spread, and [[beta-lactamase]] for drug resistance.<ref>Medical Laboratory Manual For Tropical Countries vol two</ref>