Editing Absence seizure (section)

==Cause==
An absence seizure is specifically caused by [[multifactorial inheritance]]. The [[Voltage-gated calcium channel|voltage-gated]] T-type [[T-type calcium channel|calcium channel]] is regulated by [[Gamma-aminobutyric acid receptor subunit gamma-2]] (GABRG2), [[GABRG3]], and [[CACNA1A2]] genes.<ref name="Albuja 2021"/> Inheritance of these genes is involved in the [[etiology]] (cause) of absence seizure.<ref name="Albuja 2021"/> The commonly held belief is that the genetic factor is the primary cause of childhood absence epilepsy. Furthermore, patients with childhood absence epilepsy have also been reported to exhibit certain copy number variations (CNVs), such as 15q11.2, 15q13.3, and 16p13.11 microdeletions.<ref name="Albuja 2021"/> Almost 25% of children suffering from absence seizure has a relative that suffers from seizures.<ref name=":0" /> Some specific anticonvulsant drugs such as phenytoin, carbamazepine, and vigabatrin have been identified to raise the chances of experiencing absence seizures.<ref>{{cite journal |last1=Posner |first1=Ewa |title=Absence seizures in children |journal=BMJ Clinical Evidence |date=18 December 2013 |volume=2013 |pages=0317 |pmid=24351614 |pmc=3867171 }}</ref>