Editing Cypermethrin (section)

==Human exposure==
Cypermethrin is moderately toxic through skin contact or ingestion. It may cause irritation to the skin and eyes. Symptoms of dermal exposure include numbness, tingling, itching, burning sensation, loss of bladder control, incoordination, seizures and possible death.

Pyrethroids may adversely affect the central nervous system. Human volunteers given dermal doses of 130{{nbsp}}μg/cm{{sup|2}} on the earlobe experienced local tingling and burning sensations. One man died after eating a meal cooked in a 10% cypermethrin/oil mix that was mistakenly used for cooking oil.<ref>{{Cite book|title=Pesticides and Neurological Diseases|last=Ecobichon|first=Donald J.|publisher=CRC Press|year=1993|isbn=978-0-8493-4361-2|pages=306}}</ref> Shortly after the meal, the victim experienced nausea, prolonged vomiting, stomach pains, and diarrhea which progressed to convulsions, unconsciousness and coma. Other family members exhibited milder symptoms and survived after hospital treatment.

It may cause allergic skin reactions in humans.<ref>{{cite web | url = http://pmep.cce.cornell.edu/profiles/extoxnet/carbaryl-dicrotophos/cypermet-ext.html | title = Cypermethrin | publisher = Extension Toxicology Network}}</ref> Excessive exposure can cause nausea, headache, muscle weakness, salivation, shortness of breath and seizures.

In humans, cypermethrin is deactivated by enzymatic hydrolysis to several carboxylic acid metabolites, which are eliminated in the urine. Worker exposure to the chemical can be monitored by measurement of the urinary metabolites, while severe overdosage may be confirmed by quantitation of cypermethrin in blood or plasma.<ref>R. Baselt, ''Disposition of Toxic Drugs and Chemicals in Man'', 8th edition, Biomedical Publications, Foster City, CA, 2008, pp. 389-391.</ref>