Editing Hyperphosphatemia (section)

==Causes==
{| class="wikitable"
|-
| Impaired renal phosphate excretion<ref name=Har18>Longo et al., Harrison's Principles of Internal Medicine, 18th ed., p.3089</ref> ||
* [[Renal insufficiency|Decreased kidney function]]
* [[Hypoparathyroidism|Low parathyroid hormone]]
** Developmental
** Autoimmune
** After neck surgery or radiation
** Activating mutations of the calcium-sensing receptor
* Parathyroid suppression
** Parathyroid-independent hypercalcemia
*** [[Hypervitaminosis D|Vitamin D]] or [[Hypervitaminosis A|vitamin A intoxication]]
*** [[Sarcoidosis]], other granulomatous diseases
*** Immobilization, osteolytic metastases
*** [[Milk-alkali syndrome]]
** Severe [[hypermagnesemia]] or [[hypomagnesemia]]
* [[Pseudohypoparathyroidism]]
* [[Acromegaly]]
* Tumoral calcinosis
* [[Heparin]] therapy
|-
| Massive extracellular fluid phosphate loads<ref name=Har18/> ||
* Rapid administration of exogenous phosphate (intravenous, oral, rectal)
* Extensive cellular injury or necrosis
** [[Crush injury|Crush injuries]]
** [[Rhabdomyolysis]]
** [[Hyperthermia]]
** [[Fulminant hepatitis]]
** Cytotoxic therapy
** Severe [[hemolytic anemia]]
* Transcellular phosphate shifts
** [[Metabolic acidosis]]
** [[Respiratory acidosis]]
|}
[[Hypoparathyroidism]]: In this situation, there are low levels of [[parathyroid hormone]] (PTH). PTH normally inhibits reabsorption of phosphate by the kidney. Therefore, without enough PTH there is more reabsorption of the phosphate leading to a high phosphate level in the blood.{{citation needed|date=June 2022}}

[[Chronic kidney failure]]: When the kidneys are not working well, there will be increased phosphate retention.{{citation needed|date=June 2022}}

Drugs: hyperphosphatemia can also be caused by taking oral sodium phosphate solutions prescribed for bowel preparation for [[colonoscopy]] in children.