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Retrograde amnesia
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=== Temporally graded RA === Memory loss in patients with temporally graded RA strongly follows [[Ribot's law]], meaning that one will experience more memory loss for events closer to the injury or disease onset.<ref name="Wixted_2004" /> This type of RA is commonly triggered in individuals with [[Korsakoff syndrome]] due to a combination of long-term alcohol use and [[Wernicke encephalopathy]].<ref>{{cite book | vauthors = Bauer RM, Asken B | veditors = Morgan JE, Ricker JH | chapter = Chapter 28: The Three Amnesias |date= 2008 | doi = 10.4324/9781315537511-51 | title = Textbook of Clinical Neuropsychology | edition = 2nd | pages = 678β700 | location = New York, NY | publisher = Rutlidge |isbn = 978-1-351-98599-4 }}</ref> Debate has risen about why this temporal gradient forms in the first place. Initial theories proposed that the [[hippocampus]] and [[medial temporal lobe]] are not nearly as important for long-term memories compared to short-term memories.<ref name="Lafleche_2011" /> As memory processing occurs in the brain over time, neocortical regions can directly communicate with each other, so they do not rely as heavily on the hippocampus for long-term memory storage.<ref name="Lafleche_2011" /> Therefore, if an individual experiences RA that damages the hippocampus, they will lose more short-term memories according to this theory. However, this theory has been challenged by the multiple-trace theory, which claims that the brain develops a hippocampal trace each time a memory is retrieved.<ref name="Lafleche_2011" /> Since more hippocampal traces are present for older memories, it is easier for older memories to remain intact when RA occurs.<ref name="Lafleche_2011" />
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