Editing Trans-splicing (section)

=== Oncogenesis ===
While some fusion transcripts occur via ''trans''-splicing in normal human cells,<ref name="Lei2016"/> ''trans''-splicing can also be the mechanism behind certain oncogenic [[fusion transcript]]s.<ref>{{cite journal | vauthors = Li H, Wang J, Mor G, Sklar J | title = A neoplastic gene fusion mimics trans-splicing of RNAs in normal human cells | journal = Science | volume = 321 | issue = 5894 | pages = 1357–61 | date = September 2008 | pmid = 18772439 | doi = 10.1126/science.1156725 | bibcode = 2008Sci...321.1357L | s2cid = 13605087 }}</ref><ref>{{cite journal | vauthors = Rickman DS, Pflueger D, Moss B, VanDoren VE, Chen CX, de la Taille A, Kuefer R, Tewari AK, Setlur SR, Demichelis F, Rubin MA | display-authors = 6 | title = SLC45A3-ELK4 is a novel and frequent erythroblast transformation-specific fusion transcript in prostate cancer | journal = Cancer Research | volume = 69 | issue = 7 | pages = 2734–8 | date = April 2009 | pmid = 19293179 | pmc = 4063441 | doi = 10.1158/0008-5472.CAN-08-4926 }}</ref>