Open main menu
Home
Random
Recent changes
Special pages
Community portal
Preferences
About Wikipedia
Disclaimers
Incubator escapee wiki
Search
User menu
Talk
Dark mode
Contributions
Create account
Log in
Editing
Neurotransmitter
(section)
Warning:
You are not logged in. Your IP address will be publicly visible if you make any edits. If you
log in
or
create an account
, your edits will be attributed to your username, along with other benefits.
Anti-spam check. Do
not
fill this in!
=== Glutamate === [[File:CAPON Binds Nitric Oxide Synthase, Regulating NMDA Receptor–Mediated Glutamate Neurotransmission.png|thumb|CAPON binds nitric oxide synthase, regulating NMDA receptor–mediated glutamate neurotransmission]] Furthermore, problems with producing or using glutamate have been suggestively and tentatively linked to many mental disorders, including [[autism]], [[obsessive–compulsive disorder]] (OCD), [[schizophrenia]], and [[major depressive disorder|depression]].<ref name="NIH Brain Basics">{{cite web |title=NIMH Brain Basics |url=http://www.nimh.nih.gov/health/educational-resources/brain-basics/brain-basics.shtml |publisher=[[U.S. National Institutes of Health]] |access-date=29 October 2014 |archive-date=29 October 2014 |archive-url=https://web.archive.org/web/20141029234833/http://www.nimh.nih.gov/health/educational-resources/brain-basics/brain-basics.shtml |url-status=dead }}</ref> Having too much glutamate has been linked to neurological diseases such as [[Parkinson's disease]], [[multiple sclerosis]], [[Alzheimer's disease]], [[stroke]], and [[Amyotrophic lateral sclerosis|ALS]] (amyotrophic lateral sclerosis).<ref>{{cite journal | vauthors = Bittigau P, Ikonomidou C | title = Glutamate in neurologic diseases | journal = Journal of Child Neurology | volume = 12 | issue = 8 | pages = 471–85 | date = November 1997 | pmid = 9430311 | doi = 10.1177/088307389701200802 | s2cid = 1258390 }}</ref>
Edit summary
(Briefly describe your changes)
By publishing changes, you agree to the
Terms of Use
, and you irrevocably agree to release your contribution under the
CC BY-SA 4.0 License
and the
GFDL
. You agree that a hyperlink or URL is sufficient attribution under the Creative Commons license.
Cancel
Editing help
(opens in new window)