Editing Arteritis (section)

===Types===
Arteritis may be primary or secondary to some other disease process. The primary types are:
{| class="wikitable"
|-
!colspan=3| Comparison of major types of arteritis
|-
! Arteritis !! Affected organs !! Histopathology
|-
| [[Takayasu arteritis]] || Large vessels,<ref name="pmid7909656">{{cite journal | vauthors = Kerr GS, Hallahan CW, Giordano J, Leavitt RY, Fauci AS, Rottem M, Hoffman GS | title = Takayasu arteritis | journal = Ann. Intern. Med. | volume = 120 | issue = 11 | pages = 919–29 | date = June 1994 | pmid = 7909656 | doi = 10.7326/0003-4819-120-11-199406010-00004 | s2cid = 21784938 }}</ref> including aorta and [[Aortic arch|arch]] branches<ref name=Fleshandbones>Stevens & Lowe: Pathology. At Fleshandbones.com</ref> || Histiocytes, giant cells<ref name=Fleshandbones/>
|-
| [[Giant cell arteritis]], also often called temporal arteritis (although they differ slightly) || [[Superficial temporal artery]], other medium- and large-sized vessels,<ref name=emedicine>[http://emedicine.medscape.com/article/809492-overview eMedicine Specialties > Temporal Arteritis] Author: Christopher H Lee, MD. Coauthor(s): Jean Marie Hammel, MD. Updated: Sep 8, 2009</ref> e.g. those supplying the head, eyes and [[optic nerve]]s || Lymphocytes, macrophages, and multinucleated [[giant cell]]s<ref name=emedicine/>
|-
| [[Polyarteritis nodosa]] ||Medium-sized vessels, [[central nervous system|CNS]], [[Peripheral neuropathy|PNS damage]], kidneys, [[gastrointestinal tract]], [[skeletal muscle]], heart<ref name=Fleshandbones/> || [[Neutrophil]]s, [[fibrinoid necrosis]]<ref name=Fleshandbones/>
|}

An example of a secondary arteritis is arteritis caused by infection with the fungal pathogen ''[[Candida albicans]]''.<ref>{{cite journal|last1=Nagi-Miura|first1=N|last2=Harada|first2=T|last3=Shinohara|first3=H|last4=Kurihara|first4=K|last5=Adachi|first5=Y|last6=Ishida-Okawara|first6=A|last7=Oharaseki|first7=T|last8=Takahashi|first8=K|last9=Naoe|first9=S|last10=Suzuki|first10=K|last11=Ohno|first11=N|title=Lethal and severe coronary arteritis in DBA/2 mice induced by fungal pathogen, CAWS, ''Candida albicans'' water-soluble fraction|journal=Atherosclerosis|date=June 2006|volume=186|issue=2|pages=310–320|doi=10.1016/j.atherosclerosis.2005.08.014|pmid=16157343|display-authors=3}}</ref>

====Giant cell arteritis====
{{main|Giant cell arteritis}}

Giant cell arteritis contains two different types of arteritides that are almost indistinguishable from one another.<ref name="Hollier 1–8"/>  It includes two types, temporal arteritis and Takayasu arteritis. Both types contain an occupancy of medium- and larger-sized arteries which are categorized based on the infiltration of the giant cells.<ref name="Hollier 1–8"/>

====Takayasu arteritis====

This type of arteritis is most common in females, with a median age of 25 years.<ref name="pmid7909656"/> Takayasu arteritis is more common in women of Asian descent who are in their reproductive years.<ref name="pmid7909656"/> However, over the past decades, its incidence  in Africa, Europe, and North America has been increasing.<ref name="pmid7909656"/> Takayasu arteritis is an inflammatory disease that mainly affects the larger vessels such as the aorta and its surrounding branches.<ref name="pmid7909656"/> Research focused on Takayasu arteritis in the western parts of the world remains limited. An estimation suggests that, each year, the number of cases per million people is 2.6.<ref name="pmid7909656"/>

====Temporal arteritis====

Temporal arteritis, the second type of giant cell arteritis, is also a chronic, inflammatory disease involving mid- to large-sized arteries.<ref name="Chen 333–335">{{cite journal|last=Chen|first=Chun-Hsiung|author2=Kung, Shih-Ya|author3= Tsai, Ying-Yang|author4= Liao, Hsien-Tzung|author5= Chou, Chung-Tei|author6= Huang, De-Feng|title=Temporal Arteritis|journal=Journal of the Chinese Medical Association|volume=68|issue=7|pages=333–335|doi=10.1016/S1726-4901(09)70170-4|pmid=16038374|year=2005|doi-access=}}</ref> Temporal arteritis has a higher incidence in people of Scandinavian descent.<ref name="Chen 333–335"/>  However, the incidence rate differs based on population, region and races.<ref name="Chen 333–335"/> Temporal arteritis is not uncommon in North America.<ref name="Chen 333–335"/> The incidence rate is around 0.017% for individuals over 50 years of age.<ref name="Chen 333–335"/>

Symptoms of temporal arteritis are classified as specific and nonspecific.<ref name="Chen 333–335"/>

Nonspecific symptoms:<ref name="Chen 333–335"/>
* Headache
* Low grade fever
* Sweating
* Anorexia (loss of appetite)
* Weight loss
* General malaise

Specific symptoms:<ref name="Chen 333–335"/>
* Claudication of the jaw
* Engorged, tender vessels

Specific symptoms usually develop in the advanced stages of temporal arteritis.<ref name="Chen 333–335"/> These symptoms can include damage to eyesight and sudden blindness in one or both eyes.<ref>{{Cite journal |last=Feilchenfeld |first=Zac |date=Nov 2011 |title=Answer: Can you identify this condition? |journal=[[Canadian Family Physician]] |volume=57 |issue=11 |pages=1296–1297|pmc=3215611 }}</ref> 

Polyarteritis nodosa of  unknown mechanism can cause testicular pain. It is often associated with aneurysms and Hepatitis B.