Editing Batrachotoxin (section)

==Treatment==
{{More citations needed|section|date=November 2022}}

Currently, no effective [[antidote]] exists for the treatment of batrachotoxin poisoning.<ref name=":1">{{cite book |doi=10.1016/B978-0-12-386454-3.00984-2 |chapter=Animals, Poisonous and Venomous |title=Encyclopedia of Toxicology |date=2014 |last1=Dodd-Butera |first1=T. |last2=Broderick |first2=M. |pages=246–251 |isbn=978-0-12-386455-0 }}</ref> [[Veratridine]], [[aconitine]] and [[grayanotoxin]]—like batrachotoxin—are lipid-soluble poisons which similarly alter the ion selectivity of the sodium channels, suggesting a common site of action. Due to these similarities, treatment for batrachotoxin poisoning might best be modeled after, or based on, treatments for one of these poisons. Treatment may also be modeled after that for [[digitalis]], which produces somewhat similar cardiotoxic effects.

While it is not an antidote, the membrane depolarization can be prevented or reversed by either [[tetrodotoxin]]<ref name=":1" /> (from [[puffer fish]]), which is a [[noncompetitive inhibitor]], or [[saxitoxin]].{{Citation needed|date=February 2018}} These both have effects antagonistic to those of batrachotoxin on sodium flux. Certain [[anesthetic]]s may act as [[receptor antagonist]]s to the action of this alkaloid poison, while other [[local anesthetic]]s block its action altogether by acting as [[Competitive inhibition|competitive]] antagonists.