Editing Chemoreceptor trigger zone (section)

==Antiemetic medications==
Antiemetic medications often target the CTZ to completely inhibit or greatly reduce vomiting. Most of these work by not allowing  certain blood-borne drugs (usually pain killers or [[stimulants]]) to bind to their respective receptors located in the CTZ. The antiemetic medications can block the binding site on a chemoreceptor in the CTZ, so that the emetic agent cannot bind to it to cause its emetic effects.<ref name="Porreca"/> Another way that antiemetic medications can work is by binding to a chemoreceptor in the CTZ, but instead of initiate vomiting, the medication can cause the receptors to send signals to the other parts of the vomiting center that inhibit emesis.<ref name="Porreca"/> Also, some anti-emetic medications work by lowering the amount of dopamine levels in the brain, which in turn effects how much dopamine comes in contact with dopamine receptors in the CTZ. Other antiemetic medications work similarly by lowering a different substance in the brain that is known to interact with chemoreceptors in the CTZ that cause emesis.